Resumen
This article explores the cellular and molecular intricacies of endometriosis, a gynecological condition marked by the abnormal growth of endometrium-like tissue outside the uterus. Emphasizing the roles of cyclins and cytoskeletal proteins, particularly in Epithelial?Mesenchymal Transition (EMT), the review highlights their diverse functions in cell cycle control, proliferation, evasion of apoptosis, and angiogenesis. The analysis underscores the interconnected pathways through which these proteins converge, influencing the genesis and progression of endometriosis. Understanding these complexities reveals insights into the disease?s causes and offers promise for targeted therapeutic approaches, ushering in a new era in managing this challenging disorder.