Resumen
Lipocalin-2 (LCN2) participates in multiple cellular processes, such as proliferation, survival, migration, invasion and inflammation. However, the molecular mechanisms involved in the LCN2-mediated cancer metastasis of human nasopharyngeal carcinoma remain poorly understood. Our study found that LCN2 negatively controlled cell invasion and metastasis by increasing the expression level of MET in NPC cells.